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B.D.S., 2008, D. Y. Patil Dental College, New Bombay, Maharashtra, India Advisor: Nicholas Carpino |
| Current Position | Postdoctoral Fellow, University of Utah School of Dentistry, Salt Lake City, UT |
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Research Interests |
Sts-1 and Sts-2 proteins are known regulators of immune function. By using knockout mice that lack Sts-1 and -2, I study the role these proteins play in the host response to bacterial infections. Using Francisella tularensis as a model pathogen, I hope to uncover a better understanding of host-pathogen interactions for future antibacterial therapies. |
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Publications |
Parashar, K., and Carpino, N. (2020). A role for the Sts phosphatases in negatively regulating IFN γ-mediated production of nitric oxide in monocytes. Immunity, Inflammation and Disease 8(4): 523-533. Frank, D., Naseem, S., Russo, G.., Li, C., Parashar, K., Konopka, J., and Carpino, N. (2018). Phagocytes from mice lacking the Sts phosphatases have an enhanced antifungal response to Candida albicans. MBio 9(4): e007832-18. Parashar, K., Kopping, E., Frank, D., Sampath, V., Thanassi, D.G., and Carpino, N. (2017). Increased resistance to intradermal Francisella tularensis LVS infection by inactivation of the Sts phosphatases. Infection and Immunity, 85: e00406-17. Wang, X., Parashar, K., Sitaram, A., and Bliska, J.B. (2014). The GAP activity of type III effector YopE triggers killing of Yersinia in macrophages. PLoS Pathogens 10(8): e1004346. PMID: 25165815 |
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Presentations |
Parashar, K., Kopping, E., Frank, D., Sampath, V., Thanassi, D.G., and Carpino, N. Increased Resistance to Intradermal Francisella tularensis LVS Infection by Inactivation of the Sts Phosphatases. Microbial Pathogenesis and Host Response, Cold Spring Harbor Laboratory, September 12 - 16, 2017.
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